Scleral suture fixation of a foldable intraocular lens without a scleral flap or groove.

Migraine is a prevalent and often incapacitating neurovascular disorder, affecting approximately 15-20% of the global population, with a significantly higher burden observed among women, particularly during their reproductive years []. Migraine attacks typically involve a unilateral throbbing headache, accompanied by nausea, aura, multisensory hypersensitivity (to light, sound, smell), and marked fatigue, with symptoms varying across prodrome, aura, headache, and postdrome phases []. Migraines are the second most common cause of disability globally among people under 60, according to the Global Burden of Disease Study 2024 []. Despite its widespread impact, the underlying pathophysiology of migraine remains complex and multifactorial. Key mechanisms include cortical spreading depression, which underlies aura; trigeminovascular activation, which causes neurogenic inflammation and pain; and sensory hypersensitivity due to central sensitization. Additionally, hypothalamic dysfunction, metabolic imbalance, and oxidative stress are increasingly recognized as important contributors across different migraine phases []. The link between migraine and the eye is both complex and clinically important. Migraines can affect various eye structures from the eyelids and pupils to the optic nerve and retina, often presenting as visual aura or eye pain. Many undiagnosed patients first seek eye care for symptoms such as photophobia, which can mimic other eye conditions [].