WaaL-WaaP receptor cross-talk drives phage-induced collateral sensitivity to colistin in Salmonella Pullorum.

Alzheimer's disease (AD) is the most common neurodegenerative disorder characterized by progressive cognitive decline, memory impairment, behavioral changes, and a gradual decline in the ability to perform daily activities., This disease is becoming an increasingly serious global health challenge as life expectancy increases and the population ages in various countries. According to the World Health Organization, dementia affects millions of people worldwide, with approximately 60–70% of dementia cases being caused by Alzheimer's disease., Pathologically, Alzheimer's is characterized by the accumulation of extracellular amyloid-beta plaques, the formation of neurofibrillary tangles due to hyperphosphorylation of tau protein, neuroinflammation, synaptic damage, and progressive neuronal death., Although age is the primary determinant, various studies have shown that genetic factors play a significant role in an individual's susceptibility to Alzheimer's. Advances in genomic and neurogenetic technologies over the past two decades have opened up new insights into the molecular mechanisms of this disease, particularly regarding genetic polymorphisms that play a role in increasing the risk of neurodegeneration. (See , , , , .) (See , , , , , .) Analysis of co-occurrence keywords using VOSviewer.